Neuroinflammation: A Driver of Chronic Pain States
Received Date: Nov 03, 2025 / Published Date: Nov 28, 2025
Abstract
This collection of articles explores the multifaceted role of neuroinflammation in various pain conditions. Glial cells, microglial receptors, astrocyte-derived vesicles, and peripheral immune cells are identified as key players. Mechanisms involving molecular mediators, the complement system, purinergic signaling, and neurotrophic factors are discussed. Furthermore, the impact of chemotherapy and gut microbiota dysbiosis on neuroinflammation and pain is highlighted, suggesting potential therapeutic targets for pain management
Keywords: Neuroinflammation脥戮 Pain脥戮 Glial Cells脥戮 Microglia脥戮 Astrocytes脥戮 Cytokines脥戮 Chemokines脥戮 Neuronal Hyperexcitability脥戮 Pain Sensitization脥戮 Therapeutic Targets
Citation: Lin M (2025) Neuroinflammation: A Driver of Chronic Pain States. jpar 14: 800. Doi: 10.4172/2167-0846.1000800
Copyright: 2025 Mei Lin This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution and reproduction in any medium, provided the original author and source are credited.
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